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Similarly, in in vivo experiments, Lip-1, a ferroptosis inhibitor, decreased neuronal death and neuro-inflammation in EBI by protecting mitochondrial functions and reducing lipid peroxidation, thereby ameliorating cerebral edema and neurological deficits after SAH ( 3 The Mechanism of Ferroptosis Studies have indicated that ferroptosis may be induced by different physiological conditions and pathological stress

B) Bar graph representing the percent correlation of treatment-induced postsynaptic spines to the general presynaptic marker Synapsin

Pickart L, Vasquez-Soltero JM, Margolina A (2012)