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It also supports fat loss by boosting metabolic rate, targeting visceral fat, and improving body composition (2)

Designing therapies that modulate these interactions requires not only precision but also an appreciation of the balance between protective and harmful outcomes

The entry of lipopolysaccharide into the liver via the damaged intestinal mucosal barrier stimulates the release of pro-inflammatory cytokines, and the downstream signaling pathways activation of the adaptor protein myeloid differentiation primary response 88 (MyD88) and nuclear factor-B (NF-B) have been demonstrated to be inhibited in the APS treatment group to alleviate liver steatosis (Zhang et al., 2012)

Nat Med (2018) 24(7):103646