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In addition, inhibiting METTL17 significantly reduces mitochondrial RNA methylation, impairing the translation of mitochondrial protein-coding genes, affecting mitochondrial energy metabolism, and enhancing mitochondrial and intracellular ROS and lipid peroxidation levels

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It is not yet a clinical treatment target, but it does offer a plausible mechanistic explanation for why some women with PCOS respond to broad metabolic interventions in ways that go beyond their direct mechanism of action

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