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Participants underwent pre-study baseline testing which included serum analysis of muscle damage and oxidative stress markers, maximal isometric strength, maximal muscle power and body composition analysis

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Suppression of ferroptosis in M1 macrophages may prolong their pro-inflammatory effects, which is relevant to autoimmune diseases such as multiple sclerosis (MS) and inflammatory bowel disease (IBD) (87, 88)

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The complexity of AP models makes it challenging to isolate direct mitochondrial effects from broader cellular responses

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