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Using whole-cell patch-clamp electrophysiology and micro-fluorescence Ca 2+ measurement of rat brain slices, study has demonstrated that the inhibition of hyperpolarization-activated current (Ih) determines the synaptic excitability of DA neurons

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GHK-Cu also suppresses the senescence-associated secretory phenotype (SASP): treated senescent fibroblasts show reduced secretion of IL-6, IL-8, MMP-3, PAI-1, and IGFBP-7 inflammatory mediators that drive paracrine senescence propagation and tissue dysfunction in aged organisms