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In this context, Pennacchietti and colleagues showed that HGF inhibited the antitumor effects of Met-directed TKI and mAbs even in highly sensitive MET -amplified tumor cells (51)

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peripherally (i.e., portal, superior mesenteric, lienal and inferior caval vein)) and arterial (i.e., abdominal aorta thrombosis, thus low-flow state caused by cardiac dysfunction or sever volume depletion [68])