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Pathway analyses of differential genomic occupancy for both epigenetic marks showed enrichment in several inflammatory pathways, including TNF superfamily cytokine production, IL-6-mediated signaling, LPS-mediated signaling, Toll-like receptor signaling, cytokine production, type II interferon production and chemokine-mediated signaling (Extended Data Fig

GSH is present at very high concentrations (110 mM) in humans, allowing it to scavenge ROS either directly or indirectly [15]

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In the peroxisome proliferator-activated receptor (PPAR)-induced COPD model, upregulation of GPX3 expression can reduce lung tissue damage caused by oxidative stress (Reddy et al., 2018)