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Therefore, we concluded that DDIT4/PGC-1 is important for AMSCs regulating mitochondrial function in APAP-induced hepatotoxicity and that DDIT4/PGC-1 may serve as a molecular target for the treatment of AILI

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However, its teratogenicity has been documented extensively and includes limb defects, central nervous system effects, and abnormalities of the respiratory, cardiovascular, gastrointestinal, and genitourinary systems