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Further, these data suggest that by maintaining circulating triglycerides, GSH synthesis in the liver potentially supports fat stores in the body

Mitochondrial dysfunction and alterations in metabolic pathways, such as those involving lipids, amino acids, and the TCA cycle, contribute to drug resistance, suggesting that targeting these pathways could enhance chemotherapy efficacy (Li and Li, 2021

GLP-1 RAs address this through several parallel pathways: Reduced hyperinsulinaemia less insulin circulating means less ovarian androgen stimulation

Such analyses can be performed in epidemiological studies, such as in workers exposed to lead [302, 419], or in in vitro systems which can determine the molecular basis of apoptotic effects [420,421,422,423]