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Since senescent cells may release a senescence-associated secretory phenotype, the authors suggest that lowering the senescent cell burden may reduce pro-fibrotic signaling in the surrounding cells

Collectively, these findings align with broader epidemiological data suggesting that long-term NSW may contribute to BS risk, in part via suppressed immune surveillance mechanisms 11

[22] [23] For example, motexafin gadolinium (MGd) is a new chemotherapeutic agent that selectively targets tumor cells, leading to cell death and apoptosis via inhibition of thioredoxin reductase and ribonucleotide reductase