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Thus, these data suggest that late-life targeting of IGF-1R signaling can recapitulate effects observed in genetic models of constitutive IGF-1R haploinsufficiency on lifespan

however, direct evidence in fatty liver disease remains to be substantiated

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Due to the absence of histone protection and the proximity of mitochondrial DNA (mtDNA) to sites of ROS generation, the mutation rate of mtDNA can be 10 to 100 times higher than that of nuclear DNA