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The primary mechanism of action of these drugs in the treatment of vitiligo is calcineurin inhibition, which causes the transcription of several proinflammatory cytokines essential to the pathophysiology of the early immune response to be disrupted and antigen-specific T-cell reactivity to be downregulated 26

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We confirmed these transcriptional changes in murine AKP organoid models treated with 5FU (Extended Data Fig

Dermatological and Wound Healing Research Advanced Skin Regeneration and Scar Reduction Research on individual components has shown significant effects on dermal wound healing with enhanced outcomes when anti-inflammatory control is included[14]