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Furthermore, these data contribute to understand why some ferroptosis-targeting strategies are highly efficient to reduce ATN [9, 29], whereas others highlight a disconnect between potent ferroptosis-modulating effects observed in cell culture and the comparatively modest impact of such interventions in AKI in vivo, exemplified by FSP1 deficiency [6, 9, 10]

doi: 10.1016/j.cell.2024.05.013 357 SchauwvliegheA.RijndersB

The antiangiogenic effects of IFNs are mediated by direct effects on endothelial cells and other cells in the tumor microenvironment

As shown in Table 2, the differences between normal cognitive aging and Alzheimer's disease can be subtle