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By adding a 13-amino acid N-terminal extension and substituting an arginine for glutamic acid at the third position, this analog is designed to bypass the insulin-like growth factor-binding proteins (IGFBPs) that normally inhibit IGF-1

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Inflammatory signaling is intricately linked with oxidative stress, where DAMPs released from injured ovarian cells activate TLR4 and downstream NF-B and NLRP3 inflammasome pathways