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Personalization informed by metabolic profiling, genomic insights, and biomarker monitoring may further optimize dietary response, allowing clinicians to adjust macronutrient ratios and caloric targets based on individual tolerance and treatment stage

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Recent evidence has demonstrated that aging and obesity drive maladaptive remodeling of the IFP, characterized by chronic low-grade inflammation, accumulation of senescent cells, and expression of senescence-associated secretory phenotype (SASP) factors, such as MMP-13, IL-1, and RANTES