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Second, despite certain key findings being substantiated by experimental validation, additional experiments are necessary to delve into potential mechanisms, particularly regarding the interaction between cuproptosis and ferroptosis

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Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired

Hydration, proper dosing, nutrition, and provider monitoring may help reduce unwanted effects