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Conducted via searches of PubMed, Cochrane, and Embase from database inception to June 2024, the review documented BPC-157s ability to modulate growth factor expression and enhance fibroblast migration to injury sites

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Giridharan, S

It was revealed through studies in uterine cancer models that caudatin stimulates TNFAIP1, which in turn inhibited NF-B transcriptional activity ( In vivo xenograft tumors further supported this mechanism of action, indicating that caudatin treatment of tumors led to increased levels of TNFAIP1 (Table 1, Row 11)