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This nuclear presence of FOXO4 seems to be involved in maintaining the viability of senescent cells by possibly modulating downstream senescence-associated pathways, detailed by changes in p53, Ser15-phospho-p53, and p21 protein levels

Figure 4 GGC Supplementation Reduces Metalloproteinase Activity Induced by Oligomeric A 40 Treating human brain cells with oligomeric A 40 significantly increased the activity of MMP-2 (Figure 5A) and decreased the activity of MMP-9 compared to non-treated cells (Figure 5B)