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Description

A study on uterine fibroid cells revealed that a key component of this pathway involves the overexpression of NOX4, leading to excessive ROS production [72]

however, it lacks the conserved active site serine, which has been replaced by an asparagine (66)

MET acts as a substrate for several protein tyrosine phosphatases, including the receptor PTPs density enhanced phosphatase 1 (DEP 1) and leukocyte common antigen related (LAR) and the non-receptor PTPs, PTP1B and T cell PTP [32]