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We treated cells with both iHAP1 [16], a PP2A agonist, and LB-100 [17], an inhibitor of PP2A, and showed that iHAP1 mediated the activation of PP2A mitigated hypoxia-induced neuronal death (Supplemental Fig

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This reaction is important because the H 2 O 2 might reduce the life expectancy of the erythrocytes for the increase in the speed of hemoglobin oxidation to metahemoglobin2 (Figure 1)