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Emerging therapies, including anti-IL-23 agents, TL1A inhibitors, and receptor-interacting protein kinase 1 (RIPK1) inhibitors, offer novel, targeted strategies to modulate inflammation, thereby expanding the therapeutic landscape and providing new options for patients with IBD

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GHKCu suppresses NFB (including p65 activation) and downstream cytokines such as IL6 and TNF, and simultaneously reduces reactive oxygen species

Without proper nutritional support, even the strongest healing signals from peptides have limited raw material to work with