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Consequently, its known salutary effect on gastrointestinal lesions, its effect on chronic inflammation lesions such as adjuvant arthritis and its effect as a non-steroidal anti-inflammatory agent in NSAlAs-induced gastrointestinal lesions were simultaneously studied in rats

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Our recent study demonstrates the crosstalk between impaired mitochondrial metabolism and dynamics, oxidative stress, inflammation, and ASC malfunction, which suggests that pharmacological modulation of mitochondrial activity might benefit ASC immunomodulatory potential [23]

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