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This suggests that DSIP may help protect neurons and support brain function, making it a promising candidate for therapies aimed at improving movement and coordination after stroke

but not only ions are responsible for this early BBB disruption, other mediators such as aquaporins, matrix metalloproteases (MMP), inflammatory cells, bradykinin, vascular endothelial growth factor, and nitric oxide synthase appear to have a role (76, 77)

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